Seen somewhere on the Internet (SOTI):
Category: Disaster
Quote Of The Day
From the study proving that Neil Ferguson’s Chinkvirus model contained flawed methodology (to say the least) comes this conclusion:
“On a personal level, I’d go further and suggest that all academic epidemiology be defunded. This sort of work is best done by the insurance sector. Insurers employ modellers and data scientists, but also employ managers whose job is to decide whether a model is accurate enough for real world usage and professional software engineers to ensure model software is properly tested, understandable and so on. Academic efforts don’t have these people, and the results speak for themselves.”
Hell, considering what’s come out of academia in terms of climate modeling as well as this latest fiasco, I’d prefer to have bookies produce the models, rather than universities.
And this is why charlatans like the Hockey-Stick guy (of global warming infamy) steadfastly refuse to release their code — they know it’ll fall over under the slightest scrutiny.
Had I ever tried to get this bullshit past my clients back in the day when I was involved in this kind of thing, I’d have been fired on my ass and my business cred utterly demolished. These pricks deserve no less.
Viral Load
…or, as I called it a few days back, dosage, gets a more technical examination here (the linked article, by the way, is very dense reading, but I urge everyone to plow through it anyway. You may learn something that prevents you from getting infected). A sample:
What evidence do we have that viral load matters?
Three classes of evidence seem strong.
The first is that we have a strong mechanism story we can tell. Viruses take time to multiply. When the immune system detects a virus it responds. If your initial viral load is low your immune system gets a head start, so you do better.
The second category is the terrible outcomes in health care workers on the front lines. Those who are dealing with the crisis first hand are dealing with lots of intense exposures to the virus. When they do catch it, they are experiencing high death rates. High viral load is the only theory I know about so far for why this is the case. Their cases are presumably handled at least as well as others, in terms of detection, testing, treatment and what the infected do themselves. The only other issue I can think of is that they might be reluctant to rest given how urgently their help is needed.
The third category is historical precedents.
Parents infected their children with what they hoped was exactly the minimum dose [of smallpox] required to get them sick enough to develop antibodies and gain immunity. Sometimes this went wrong and the child would get sick. Thus this form of inoculation was dangerous and 1%-2% of patients died. But of those who got smallpox infections in other ways, 20%-30% of patients died. Those rates are well established.
I should point out that Doc Russia, who as an ER doctor has been treating Chinkvirus patients almost daily, fully expected to catch the virus himself, but so far [crossing fingers] hasn’t. All I can think of is that because his hospitals (he works in several) don’t have that many infectees compared to those in, say, London or New York, his aggregate exposure is low; that, his age outside the at-risk group, plus his fanatical adherence to commonsense protective measures, has probably kept him well. Which leads to the other major point in the above linked article:
The default model is that the longer and more closely you interact with an infected person, especially a symptomatic infected person, the larger your viral load.
In-household infections are presumed to be high viral load, as in the case of measles. So would be catching the infection while treating patients.
Most out-of-household infections that aren’t health care related are presumed to be low viral load. Anything outdoors is probably low viral load. Most methods that involve surfaces are probably low viral load. Infection via the air from someone there half an hour ago, to the extent this is a thing, is low viral load. Quick interactions with asymptomatic individuals are probably low viral load.
I should point out that the above are observations based on admittedly-poor data, but as we know that the level of dosage/viral load is critical in other diseases (measles, smallpox, SARS etc.), it’s not a bad deduction to assume that it’s true also of the Chinkvirus.
As with all decisions in life, the key to decision-making is risk assessment and odds-calculation. Use all the above accordingly, as you plan your daily life.
Stupid People
One of the most unattractive things that has come out of the Chinkvirus pandemic has been the social shaming of people who, in the opinions of some, are ignoring the dangers of the virus’s spread. Brits have coined a term “covidiots” to describe these people, hence (link in pic):
Well of course they would be fearful, because — and let’s be under no illusions about this — when it comes to viral infection, only two things matter: dosage (the actual number of viruses inhaled or ingested) and its subset, dispersion.
Most studies on infection take place in a closed room of about 400 sq.ft. (20′ x 20′). Now take that outside (especially on a breezy day), and the dosage will be immediately reduced to an enormous degree because the wind not only disperses the virus-laden particles, but can even blow them apart, reducing their danger exponentially. It’s why the Nazis went to all the trouble of building gas chambers at Auschwitz, instead of just spraying Zyklon-B on the hapless Jews out in the fields.
So to return to the above hysteria: of course a majority of people are going to be apprehensive about going back to the office — it’s a closed environment, you idiots, and viral infection is definitely a possibility. But out in the open air?
Nada, zip, zilch — as long as people keep some distance between themselves and strangers so that the open air can work its magic. And don’t touch railings and other surfaces that others have touched without cleaning your hands with disinfectant wipes immediately afterwards.
And as for those idiot cops who keep harassing sunbathers, surfers and the like: the cops should be tied to lampposts and hosed down with icy water (lest they get viral infections by getting too close to the people they’re harassing), e.g.:
And those moron journalists [redundancy alert] who perpetuate this foolishness deserve the same treatment.
Silver Lining
If anything good has come out of the Chinkvirus pandemic, it’s this:
A comprehensive study of behaviours and attitudes since the outbreak began found that three in five people will stop greeting friends with a hug and a kiss, and will also avoid crowded places in the future.
Include me in that number, although I hardly ever did it anyway. This modern thing of men hugging other men who are not family has always given me the heebies. I hug my son — and not even that often — and occasionally my friend Trevor (who insists on doing it because he knows it bugs me, and I don’t kill him because he’s my friend). Other than that, ugh.
I don’t mind shaking hands, however, because I was brought up to do that with men, further affection being communicated by a punch or slap on the shoulder.
But not with women. Unless it’s a business thing, I’m always tempted to turn a handshake with a woman into kissing her hand; mostly, it’s greeted with giggles and sighs. If I add, “Sorry, but I was brought up to love and respect women,” the response is universally positive. Hugging is too intimate; kissing a hand denotes respect.
As for hugging and kissing women I know… well, I’m never going to stop doing that. (At the doctor’s the other day, I complained to his nurse practitioner — whom I’ve known for over fifteen years — that I wasn’t going to molest her as I usually do when I visit. She shook her head sadly and said, “And I always look so forward to it, too.” Aaah, Texas.)
Ultimately, though, I think that for the next few years we as a society are going to be more comfortable about keeping other people — and certainly strangers — at arm’s length, so to speak. And that’s a Good Thing. But as time passes, we’ll forget all about pandemic behavior and relapse into over-familiarity, which isn’t.
Over-Complicating
I have often snarled at the .dotmil, who never seem to miss an opportunity to create weapons systems that are so laden with bells and whistles that they add all sorts of other problems, e.g. COST not to mention MORE THINGS TO BREAK.
It’s not just the military, of course. Try this wonderful screw-up from a different government department (emphasis added):
There’s a massive shortage of COVID-19 (Coronavirus) test kits in the U.S., as cases continue to skyrocket in places like Seattle and New York City. This is largely due to the failure of the Centers for Disease Control and Prevention (CDC) to distribute the tests in a timely fashion.
But it didn’t have to be this way. Back in January and February—when cases of the deadly disease began aggressively circulating outside of China—diagnostics already existed in places like Wuhan, where the pandemic began. Those tests followed World Health Organization (WHO) test guidelines, which the U.S. decided to eschew.
Instead, the CDC created its own in-depth diagnostics that could identify not only COVID-19, but a host of SARS-like coronaviruses.
“No, not just a test kit to address the immediate issue; let’s make one that’s more complicated but can test for every single virus in the world, plus others that don’t even exist yet!”
And as any fule kno, when you try to make something to do one thing, but expand the mission for it to do lots of things…
Then, disaster struck: When the CDC sent tests to labs during the first week of February, those labs discovered that while the kits did detect COVID-19, they also produced false positives when checking for other viruses. As the CDC went back to the drawing board to develop yet more tests, precious time ticked away.
Government: fucking it up six ways to Sunday, each and every time.